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The activity of the mitochondrial enzyme, glycine decarboxylase (GDC), is sensitive to oxidative stress (Taylor et al., 2002), and this enzyme has recently been shown to be inhibited by thiol modifications on several cysteine residues (Palmieri et al., 2010)

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To verifiy the blood pressure-lowering effect of the eNOS/NO pathway, a specific inhibitor Nω-Nitro-L-arginine methyl ester (L-NAME) was used to block eNOS activity, which significantly eliminated the GA-mediated beneficial effects, such as the reduction in SBP, aortic thickening, and collagen deposition (Yan et al., 2020)